Publikationer av Jacob Odeberg
Refereegranskade
Artiklar
[1]
S. Öling et al., "A human stomach cell type transcriptome atlas," BMC Biology, vol. 22, no. 1, 2024.
[2]
E. C. Struck et al., "Global Transcriptome Analysis Reveals Distinct Phases of the Endothelial Response to TNF," Journal of Immunology, vol. 212, no. 1, s. 117-129, 2024.
[3]
R. V. Diaz et al., "Plasma levels of complement components C5 and C9 are associated with thrombin generation," Journal of Thrombosis and Haemostasis, vol. 22, no. 9, s. 2531-2542, 2024.
[4]
M. Chemaly et al., "Biliverdin Reductase B Is a Plasma Biomarker for Intraplaque Hemorrhage and a Predictor of Ischemic Stroke in Patients with Symptomatic Carotid Atherosclerosis," Biomolecules, vol. 13, no. 6, 2023.
[5]
M. J. Iglesias et al., "Elevated plasma complement factor H related 5 protein is associated with venous thromboembolism," Nature Communications, vol. 14, no. 1, 2023.
[6]
H. Englert et al., "Targeting NETs using dual-active DNase1 variants," Frontiers in Immunology, vol. 14, 2023.
[7]
M. Norreen-Thorsen et al., "A human adipose tissue cell-type transcriptome atlas," Cell Reports, vol. 40, no. 2, 2022.
[8]
P. Smith et al., "Markers of neutrophil activation and neutrophil extracellular traps in diagnosing patients with acute venous thromboembolism : A feasibility study based on two VTE cohorts," PLOS ONE, vol. 17, no. 7 July, 2022.
[9]
F. Edfors et al., "Proteomics in thrombosis research," RESEARCH AND PRACTICE IN THROMBOSIS AND HAEMOSTASIS, vol. 6, no. 3, 2022.
[10]
O. Bocher et al., "Testing for association with rare variants in the coding and non-coding genome : RAVA-FIRST, a new approach based on CADD deleteriousness score," PLOS Genetics, vol. 18, no. 9, s. e1009923, 2022.
[11]
B. E. Suur et al., "Therapeutic potential of the Proprotein Convertase Subtilisin/Kexin family in vascular disease," Frontiers in Pharmacology, vol. 13, 2022.
[12]
M. Karlsson et al., "A single-cell type transcriptomics map of human tissues," Science Advances, vol. 7, no. 31, 2021.
[13]
M. Razzaq et al., "An artificial neural network approach integrating plasma proteomics and genetic data identifies PLXNA4 as a new susceptibility locus for pulmonary embolism," Scientific Reports, vol. 11, no. 1, 2021.
[14]
M. J. Iglesias et al., "Identification of Endothelial Proteins in Plasma Associated With Cardiovascular Risk Factors," Arteriosclerosis, Thrombosis and Vascular Biology, vol. 41, no. 12, s. 2990-3004, 2021.
[15]
T. Dodig-Crnkovic et al., "Facets of individual-specific health signatures determined from longitudinal plasma proteome profiling," EBioMedicine, vol. 57, 2020.
[16]
T. Abdellah et al., "Integration of molecular profiles in a longitudinal wellness profiling cohort," Nature Communications, vol. 11, no. 1, 2020.
[17]
U. Rykaczewska et al., "PCSK6 Is a Key Protease in the Control of Smooth Muscle Cell Function in Vascular Remodeling," Circulation Research, vol. 126, no. 5, s. 571-585, 2020.
[18]
M.-G. Hong et al., "Profiles of histidine-rich glycoprotein associate with age and risk of all-cause mortality," Life Science Alliance, vol. 3, no. 10, s. e202000817, 2020.
[19]
S. Röhl et al., "Transcriptomic profiling of experimental arterial injury reveals new mechanisms and temporal dynamics in vascular healing response," JVS-Vascular Science, vol. 1, s. 13-27, 2020.
[20]
P. Dusart et al., "A Systems-Based Map of Human Brain Cell-Type Enriched Genes and Malignancy-Associated Endothelial Changes," Cell Reports, vol. 29, no. 6, s. 1690-+, 2019.
[21]
M. Uhlén et al., "A genome-wide transcriptomic analysis of protein-coding genes in human blood cells," Science, vol. 366, no. 6472, s. 1471-+, 2019.
[22]
E. Karlöf et al., "Correlation of computed tomography with carotid plaque transcriptomes associates calcification with lesion-stabilization," Atherosclerosis, vol. 288, s. 175-185, 2019.
[23]
J. Odeberg et al., "A novel cysteine-linked antibacterial surface coating significantly inhibits bacterial colonization of nasal silicone prongs in a phase one pre-clinical trial," Materials science & engineering. C, biomimetic materials, sensors and systems, vol. 93, s. 782-789, 2018.
[24]
P. Dusart et al., "A systems-approach reveals human nestin is an endothelial-enriched, angiogenesis-independent intermediate filament protein," Scientific Reports, vol. 8, no. 1, 2018.
[25]
L. P. Matic et al., "Novel Multiomics Profiling of Human Carotid Atherosclerotic Plaques and Plasma Reveals Biliverdin Reductase B as a Marker of Intraplaque Hemorrhage," JACC: Basic to Translational Science, vol. 3, no. 4, s. 464-480, 2018.
[26]
A. Majeed et al., "Management of rivaroxaban- or apixaban-associated major bleeding with prothrombin complex concentrates : a cohort study," Blood, vol. 130, no. 15, s. 1706-1712, 2017.
[27]
L. M. Butler et al., "Analysis of Body-wide Unfractionated Tissue Data to Identify a Core Human Endothelial Transcriptome," Cell Systems, vol. 3, no. 3, s. 287-301.e3, 2016.
[28]
M. Ljungqvist et al., "Cardiovascular disease and mortality after a first episode of venous thromboembolism in young and middle-aged women," Thrombosis Research, vol. 138, s. 80-85, 2016.
[29]
M. Bruzelius et al., "F11 is associated with recurrent VTE in women A prospective cohort study," Thrombosis and Haemostasis, vol. 115, no. 2, s. 406-414, 2016.
[30]
L. Perisic et al., "Gene expression signatures, pathways and networks in carotid atherosclerosis," Journal of Internal Medicine, vol. 279, no. 3, s. 293-308, 2016.
[31]
D. Edsgärd et al., "GeneiASE : Detection of condition-dependent and static allele-specific expression from RNA-seq data without haplotype information," Scientific Reports, vol. 6, 2016.
[32]
J. Odeberg et al., "Influence of pre-existing inflammation on the outcome of acute coronary syndrome : a cross-sectional study," BMJ Open, vol. 6, no. 1, 2016.
[33]
M. Bruzelius et al., "PDGFB, a new candidate plasma biomarker for venous thromboembolism : Results from the VEREMA affinity proteomics study," Blood, vol. 128, no. 23, s. e59-e66, 2016.
[34]
M. J. Iglesias et al., "An affinity proteomics study for plasma biomarker candidates of cardiovascular disease in venous thromboembolism," Journal of Thrombosis and Haemostasis, vol. 13, s. 956-956, 2015.
[35]
M. Bruzelius et al., "F11 is associated with recurrent event of VTE in women : a prospective cohort study," Journal of Thrombosis and Haemostasis, vol. 13, s. 198-198, 2015.
[36]
M. Bruzelius et al., "Predicting venous thrombosis in women using a combination of genetic markers and clinical risk factors," Journal of Thrombosis and Haemostasis, vol. 13, no. 2, s. 219-227, 2015.
[37]
M. Uhlén et al., "Tissue-based map of the human proteome," Science, vol. 347, no. 6220, s. 1260419, 2015.
[38]
M. Bruzelius et al., "Verema - an affinity proteomics study to identify and translate plasma biomarkers for venous thromboembolism," Journal of Thrombosis and Haemostasis, vol. 13, s. 954-954, 2015.
[39]
L. Fagerberg et al., "Analysis of the human tissue-specific expression by genome-wide integration of transcriptomics and antibody-based proteomics," Molecular & Cellular Proteomics, vol. 13, no. 2, s. 397-406, 2014.
[40]
M. Bruzelius et al., "Influence of coronary artery disease-associated genetic variants on risk of venous thromboembolism," Thrombosis Research, vol. 134, no. 2, s. 426-432, 2014.
[41]
D. A. Liem et al., "Molecular- and Organelle-Based Predictive Paradigm Underlying Recovery by Left Ventricular Assist Device Support," Circulation Heart Failure, vol. 7, no. 2, s. 359-366, 2014.
[42]
M. Habuka et al., "The Kidney Transcriptome and Proteome Defined by Transcriptomics and Antibody-Based Profiling," PLOS ONE, vol. 9, no. 12, s. e116125, 2014.
[43]
J. Odeberg et al., "The influence of smoking and impaired glucose homoeostasis on the outcome in patients presenting with an acute coronary syndrome : a cross-sectional study," BMJ Open, vol. 4, no. 7, s. e005077, 2014.
[44]
L. Fagerberg et al., "Contribution of antibody-based protein profiling to the human chromosome-centric proteome project (C-HPP)," Journal of Proteome Research, vol. 12, no. 6, s. 2439-2448, 2013.
[45]
A. Li et al., "Heart research advances using database search engines, human protein atlas and the sydney heart bank," Heart, Lung and Circulation, vol. 22, no. 10, s. 819-826, 2013.
[46]
N. C. Zong et al., "Integration of cardiac proteome biology and medicine by a specialized knowledgebase," Circulation Research, vol. 113, no. 9, s. 1043-1053, 2013.
[47]
L. Perisic et al., "Profiling of atherosclerotic lesions by gene and tissue microarrays reveals pcsk6 as a novel protease in unstable carotid atherosclerosis," Arteriosclerosis, Thrombosis and Vascular Biology, vol. 33, no. 10, s. 2432-2443, 2013.
[48]
U. Qundos et al., "Profiling post-centrifugation delay of serum and plasma with antibody bead arrays," Journal of Proteomics, vol. 95, no. SI, s. 46-54, 2013.
[49]
M. Uhlén et al., "Antibody-based Protein Profiling of the Human Chromosome 21," Molecular & Cellular Proteomics, vol. 11, no. 3, 2012.
[50]
M. J. Iglesias et al., "Combined Chromatin and Expression Analysis Reveals Specific Regulatory Mechanisms within Cytokine Genes in the Macrophage Early Immune Response," PLOS ONE, vol. 7, no. 2, s. e32306, 2012.
[51]
A. Sillén et al., "Linkage Analysis of Autopsy-Confirmed Familial Alzheimer Disease Supports an Alzheimer Disease Locus in 8q24," Dementia and Geriatric Cognitive Disorders, vol. 31, no. 2, s. 109-118, 2011.
[52]
A. Sillén et al., "Linkage to 20p13 including the ANGPT4 gene in families with mixed Alzheimer's disease and vascular dementia," Journal of Human Genetics, vol. 55, no. 10, s. 649-655, 2010.
[53]
C. B. Estigoy et al., "Intercalated discs : Multiple proteins perform multiple functions in non-failing and failing human hearts," Biophysical Reviews, vol. 1, no. 1, s. 43-49, 2009.
[54]
A. Sillén et al., "Expanded high-resolution genetic study of 109 Swedish families with Alzheimer's disease," European Journal of Human Genetics, vol. 16, no. 2, s. 202-208, 2008.
[55]
M. Andersen et al., "In silico detection of sequence variations modifying transcriptional regulation," PloS Computational Biology, vol. 4, no. 1, s. e5, 2008.
[56]
J. Odeberg et al., "The Asp(298) allele of endothelial nitric oxide synthase is a risk factor for myocardial infarction among patients with type 2 diabetes mellitus," BMC Cardiovascular Disorders, vol. 8, 2008.
[57]
L. Berglund et al., "The epitope space of the human proteome," Protein Science, vol. 17, no. 4, s. 606-613, 2008.
[58]
M. van Rooijen et al., "APC resistance during the normal menstrual cycle," Thrombosis and Haemostasis, vol. 98, no. 6, s. 1246-1251, 2007.
[59]
L. Cheung et al., "Hormonal and nutritional regulation of alternative CD36 transcripts in rat liver : a role for growth hormone in alternative exon usage," BMC Molecular Biology, vol. 8, no. 60, s. 12, 2007.
[60]
J. Andrade et al., "The use of grid computing to drive data-intensive genetic research," European Journal of Human Genetics, vol. 15, no. 6, s. 694-702, 2007.
[61]
P. Lahermo et al., "A quality assessment survey of SNP genotyping laboratories," Human Mutation, vol. 27, no. 7, s. 711-714, 2006.
[62]
C. Zhu et al., "Allele-specific MMP-3 transcription under in vivo conditions," Biochemical and Biophysical Research Communications - BBRC, vol. 348, no. 3, s. 1150-1156, 2006.
[63]
M. Andersen et al., "Alternative promoter usage of the membrane glycoprotein CD36," BMC Molecular Biology, vol. 7, s. 8, 2006.
[64]
M. Käller et al., "Comparison of PrASE and Pyrosequencing for SNP Genotyping," BMC Genomics, vol. 7, s. 291, 2006.
[65]
J. Odeberg et al., "Severity of acute coronary syndrome is predicted by interactions between fibrinogen concentrations and polymorphisms in the GPIIIa and FXIII genes," Journal of Thrombosis and Haemostasis, vol. 4, no. 4, s. 909-912, 2006.
[66]
J. M. Odeberg et al., "UGT1A polymorphisms in a Swedish cohort and a human diversity panel, and the relation to bilirubin plasma levels in males and females," European Journal of Clinical Pharmacology, vol. 62, no. 10, s. 829-837, 2006.
[67]
J. Andrade et al., "Using Grid Technology for Computationally Intensive Applied Bioinformatics Analyses," In Silico Biology, vol. 6, no. 6, s. 495-504, 2006.
[68]
T. Sandalova et al., "A structural basis for CD8(+) T cell-dependent recognition of non-homologous peptide ligands - Implications for molecular mimicry in autoreactivity," Journal of Biological Chemistry, vol. 280, no. 29, s. 27069-27075, 2005.
[69]
D. Velazquez-Fernandez et al., "Expression profiling of adrenocortical neoplasms suggests a molecular signature of malignancy.," Surgery, vol. 138, no. 6, s. 1087-1094, 2005.
[70]
K. Holmberg et al., "Pyrosequencing analysis of thrombosis-associated risk markers," Clinical Chemistry, vol. 51, no. 8, s. 1549-1552, 2005.
[71]
V. Magnusson et al., "Both risk alleles for Fc gamma RIIA and Fc gamma RIIIA are susceptibility factors for SLE : a unifying hypothesis," Genes and Immunity, vol. 5, no. 2, s. 130-137, 2004.
[72]
V. Magnusson et al., "Polymorphisms of the Fc gamma receptor type IIB gene are not associated with systemic lupus erythematosus in the Swedish population," Arthritis and Rheumatism, vol. 50, no. 4, s. 1348-1350, 2004.
[73]
A. Lindstrom, J. Odeberg och J. Albert, "Pyrosequencing for detection of lamivudine-resistant hepatitis B virus," Journal of Clinical Microbiology, vol. 42, no. 10, s. 4788-4795, 2004.
[74]
S. Boräng et al., "Vascular gene expression in atherosclerotic plaque-prone regions analyzed by representational difference analysis," Pathobiology (Basel), vol. 71, no. 2, s. 107-114, 2004.
[75]
T. Andersson et al., "Shotgun sequencing and microarray analysis of RDA transcripts," Gene, vol. 310, s. 39-47, 2003.
[76]
C. Agaton et al., "Gene expression analysis by signature pyrosequencing," Gene, vol. 289, no. 1-2, s. 31-39, 2002.
[77]
J. Odeberg et al., "Molecular haplotyping by pyrosequencing (TM)," BioTechniques, vol. 33, no. 5, s. 1104-+, 2002.
[78]
T. Andersson et al., "Monitoring of representational difference analysis subtraction procedures by global microarrays," BioTechniques, vol. 32, no. 6, s. 1348-+, 2002.
[79]
D. O'Meara et al., "SNP typing by apyrase-mediated allele-specific primer extension on DNA microarrays," Nucleic Acids Research, vol. 30, no. 15, 2002.
[80]
A. Ahmadian et al., "Genotyping by apyrase-mediated allele-specific extension," Nucleic Acids Research, vol. 29, no. 24, 2001.
[81]
S. Borang et al., "Monitoring of the subtraction process in solid-phase representational difference analysis : characterization of a candidate drug," Gene, vol. 271, no. 2, s. 183-192, 2001.
[82]
T. Andersson et al., "Novel candidate genes for atherosclerosis are identified by representational difference analysis-based transcript profiling of cholesterol-loaded macrophages," Pathobiology (Basel), vol. 69, no. 6, s. 304-314, 2001.
[83]
J. Odeberg et al., "A cDNA RDA protocol using solid-phase technology suited for analysis in small tissue samples.," Biomolecular Engineering, vol. 17, no. 1, s. 1-9, 2000.
[84]
P. Tollet-Egnell et al., "Differential cloning of growth hormone-regulated hepatic transcripts in the aged rat," Endocrinology, vol. 141, no. 3, s. 910-921, 2000.
[85]
O. Rosok et al., "The C1orf9 gene encodes a putative transmembrane member of a novel protein family," Biochemical and Biophysical Research Communications - BBRC, vol. 267, no. 3, s. 855-862, 2000.
[86]
J. Odeberg et al., "Cloning and characterization of ZNF189, a novel human Krüppel-like zinc finger gene localized to chromosome 9q22-q31.," Genomics, vol. 50, no. 2, s. 213-21, 1998.
[87]
A. Ahmadian et al., "Genetic instability in the 9q22.3 region is a late event in the development of squamous cell carcinoma.," Oncogene, vol. 17, no. 14, 1998.
[88]
O. Røsok et al., "Solid-phase method for differential display of genes expressed in hematopoietic stem cells.," BioTechniques, vol. 21, no. 1, s. 114-21, 1996.
[89]
G. H. Gudmundsson et al., "The human gene FALL39 and processing of the cathelin precursor to the antibacterial peptide LL-37 in granulocytes.," European Journal of Biochemistry, vol. 238, no. 2, s. 325-32, 1996.
[90]
B. Agerberth et al., "FALL-39, a putative human peptide antibiotic, is cysteine-free and expressed in bone marrow and testis.," Proceedings of the National Academy of Sciences of the United States of America, vol. 92, no. 1, s. 195-9, 1995.
Konferensbidrag
[91]
M. Razzaq et al., "Explainable Artificial Neural Network for Recurrent Venous Thromboembolism Based on Plasma Proteomics," i Computational Methods in Systems Biology19th International Conference, CMSB 2021, Bordeaux, France, September 22–24, 2021, Proceedings, 2021, s. 108-121.
[92]
H. Lorenzo et al., "High-dimensional multi-block analysis of factors associated with thrombin generation potential," i Proceedings 2019 IEEE 32ND INTERNATIONAL SYMPOSIUM ON COMPUTER-BASED MEDICAL SYSTEMS (CBMS), 2019, s. 453-458.
[93]
J. Andrade et al., "Applications of grid computing in genetics and proteomics," i Applied Parallel Computing : State Of The Art In Scientific Computing, 2007, s. 791-798.
[94]
T. Sandholm et al., "Market-Based Resource Allocation using Price Prediction in a high performance computing Grid for scientific applications," i Proceedings of the IEEE International Symposium on High Performance Distributed Computing 2006, 2006, s. 132-143.
Kapitel i böcker
[95]
M. J. Iglesias, J. M. Schwenk och J. Odeberg, "Affinity Proteomics Assays for Cardiovascular and Atherosclerotic Disease Biomarkers," i Protein Microarrays for Disease Analysis : Methods and Protocols, : Springer Nature, 2021, s. 163-179.
Icke refereegranskade
Artiklar
[96]
M. Chemaly et al., "Biliverdin Reductase B is a Plasma Biomarker for Intraplaque Hemorrhage and A Predictor of Ischemic Stroke in Symptomatic Carotid Stenosis," Atherosclerosis, vol. 379, s. S180-S180, 2023.
[97]
C. G. dos Remedios et al., "Proteomics of the Human Cardiac Intercalated Disc : A More Complex Multi-Functional Structure than was Previously Thought," Biophysical Journal, vol. 98, no. 3, s. 755A-756A, 2010.
Avhandlingar
[98]
J. Odeberg, "Analysis of human gene transcripts : Identification and characterisation based on homology and differential expression," Doktorsavhandling Stockholm : KTH, 1998.
Övriga
[99]
J. Odeberg et al., "Direct sequencing of the ZNF 189 gene promotor reveals artifact hot spot mutations.," (Manuskript).
[100]
Senaste synkning med DiVA:
2024-11-18 00:13:24